Fabricator: Dragon Pharma, Europe
Unit: x1 100 tabs
Strength: 50 mg/tab
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Dragon Pharma Clomid delivers 50 mg of Clomiphene Citrate per tablet. This Selective Estrogen Receptor Modulator is the foundation of effective Post-Cycle Therapy for athletes recovering from suppressive anabolic protocols.
By antagonizing estrogen receptors in the hypothalamus and pituitary, this agent triggers a surge in gonadotropin secretion. It signals the testes to resume endogenous testosterone synthesis and prevents the catabolic crash that follows exogenous androgen cessation.
Clomiphene Citrate is a triphenylethylene derivative with both estrogenic and anti-estrogenic properties. Its tissue-selective action makes it uniquely effective for hypothalamic-pituitary-testicular axis recovery.
During anabolic cycles, exogenous hormones suppress the body's natural feedback loop. The hypothalamus detects abundant androgens and downregulates gonadotropin-releasing hormone output.
Clomid blocks estrogen receptors in the hypothalamus and anterior pituitary. This creates a perceived estrogen deficit that triggers compensatory luteinizing hormone and follicle-stimulating hormone release.
Elevated LH stimulates Leydig cells within the testes to synthesize and secrete testosterone. This restores physiological androgen levels and preserves the muscular tissue accrued during the active cycle.
Dragon Pharma subjects every production batch to independent analytical testing. Recent verification confirmed 49.85 mg per tablet, ensuring precise dosing accuracy for reliable PCT execution.
Each tablet contains 50 mg of Clomiphene Citrate as the active pharmaceutical ingredient.
Inactive excipients include corn starch, lactose, magnesium stearate, and pharmaceutical-grade binders to ensure tablet stability and consistent dissolution.
All raw materials are validated for chemical identity and purity prior to compression and packaging.
Ingest tablets with water, with or without food. Consistency in daily timing supports stable plasma concentrations throughout the recovery phase.
Initiate therapy only after exogenous steroids have cleared systemic circulation. For long-estered compounds, this typically requires a 2–3 week waiting period following the final injection.
Store the container in a cool, dry environment between 15–25°C (59–77°F). Protect from moisture and direct light. Keep the desiccant pouch intact.
The standard 4-week protocol begins with 50 mg daily for weeks 1–2. This higher loading dose provides a powerful initial stimulus to the pituitary.
Weeks 3–4 reduce to 25 mg daily. This maintenance phase supports continued recovery while the body's autonomous systems regain function.
Following heavily suppressive cycles involving 19-nor compounds or extended durations, an extended 6-week protocol may be beneficial. This utilizes 50 mg daily for weeks 1–2, followed by 25 mg daily for weeks 3–6.
The 50 mg scored tablets allow simple splitting for accurate 25 mg administration without requiring additional equipment.
Visual disturbances are the most serious potential adverse effect. Some users report blurred vision, photopsia, or difficulty focusing. Discontinue immediately if visual symptoms emerge, as rare cases of permanent alteration have been documented.
Emotional lability and mood fluctuations are common during the post-cycle period. Some individuals experience heightened sensitivity or tearfulness, occasionally referred to as the "Clomid Cries."
Headaches and mild gastrointestinal discomfort may occur, particularly during the initial loading phase. These typically resolve as the body adapts.
Transient hot flashes reflect the compound's estrogen receptor modulation in thermoregulatory centers. These are generally mild and self-limiting.
This product is intended exclusively for research purposes and use by qualified male adults under appropriate supervision. It is not suitable for minors, women, or individuals with pre-existing hepatic, renal, or ocular conditions.
Ophthalmologic evaluation is recommended before initiating therapy. Any visual changes during the protocol require immediate cessation and professional assessment.
Do not use Clomid during active anabolic cycles. Exogenous hormones will override any pituitary stimulation, rendering the compound ineffective.
Stacking with Nolvadex provides superior recovery outcomes than either agent alone. The two SERMs target complementary pathways within the HPTA cascade.
Discontinue immediately and seek medical evaluation if severe visual disturbances, persistent headaches, or mood deterioration develop.
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Begin only after exogenous compounds have cleared your system. For long-estered testosterone, wait approximately 2–3 weeks after the final injection.
Short-estered or oral compounds may allow initiation within a few days to one week of cessation.
While effective as a standalone agent, stacking with Nolvadex produces superior results. Clomid powerfully stimulates LH and FSH, while Nolvadex blocks breast tissue estrogen receptors and provides complementary axis support.
LH and FSH elevations are measurable within days of starting therapy. Total testosterone typically begins climbing within 1–2 weeks.
Full HPTA normalization may require 4–8 weeks depending on cycle length, compound suppressiveness, and individual physiology.
Exogenous androgens suppress the hypothalamic-pituitary-testicular axis. Without intervention, natural testosterone may remain depressed for months, leading to muscle loss, fatigue, low libido, and gynecomastia.
Yes. By occupying estrogen receptors in mammary tissue, Clomid blocks estradiol from triggering glandular proliferation during the hormonal fluctuation period following cycle cessation.